What causes somatic symptom disorder?

Somatic Symptom Disorder (SSD) arises from a complex interplay of psychological, biological, and social factors that cause individuals to experience persistent and distressing physical symptoms without a fully explainable medical cause. At its core, SSD involves the mind and body being deeply connected in a way that physical sensations are amplified or misinterpreted, leading to real and often debilitating symptoms such as pain, fatigue, dizziness, or shortness of breath.

One major cause is **heightened body awareness**. People with SSD tend to focus intensely on normal bodily sensations or minor discomforts, which are then perceived as severe or threatening. This excessive attention can amplify the sensation, making it feel more intense and distressing than it objectively is. The brain’s interpretation of these signals becomes skewed, creating a feedback loop where anxiety about symptoms increases the physical sensations, which in turn fuels more anxiety.

**Stress and anxiety** play a crucial role. Chronic stress or anxiety, especially when related to health concerns, can trigger or worsen somatic symptoms. The body’s stress response can produce physical effects like muscle tension, increased heart rate, or gastrointestinal upset, which may be misread as signs of serious illness. Over time, this can develop into persistent symptoms characteristic of SSD. The mind’s inability to regulate these stress responses effectively contributes to ongoing symptom distress.

**Trauma**, particularly early life trauma such as childhood physical or sexual abuse, is another significant factor. Trauma can disrupt normal emotional processing and lead to somatization, where emotional pain is expressed through physical symptoms. The body essentially becomes a vessel for unprocessed psychological distress, manifesting symptoms that are very real to the person but lack a clear medical explanation.

Biological factors also contribute. Some individuals may have a genetic predisposition or neurobiological differences that affect how they perceive and process pain or bodily sensations. For example, altered functioning in brain areas responsible for sensory processing and emotional regulation can make someone more vulnerable to developing SSD.

Social and environmental influences matter as well. Growing up in an environment where illness was heavily emphasized or where emotional expression was discouraged can lead to a tendency to express distress physically rather than verbally. Additionally, repeated medical testing and reassurance without clear answers can reinforce symptom focus and health anxiety, perpetuating the disorder.

Another important aspect is the **interaction between psychological and physical health**. The body and mind continuously influence each other. When a person experiences a physical sensation, the brain interprets it based on past experiences, current emotional state, and cognitive biases. If the interpretation is negative or catastrophic, it can heighten symptom perception and distress. This cycle of symptom amplification and anxiety can become self-sustaining.

In some cases, somatic symptoms may also be linked to **functional neurological disorder (FND)**, which shares similarities with SSD. Both involve symptoms that are involuntary and not explained by traditional medical conditions, but FND typically involves neurological symptoms like weakness or sensory changes. The overlap suggests that disruptions in brain networks that integrate sensory input and emotional processing are central to these disorders.

It is important to note that the symptoms in SSD are genuine and not fabricated or intentionally produced. The disorder is not about faking illness but about how the brain and body communicate distress. This makes diagnosis challenging, as medical tests often fail to find a clear cause, leading to frustration for both patients and clinicians.

In summary, somatic symptom disorder is caused by a combination of heightened bodily awareness, psychological stress and anxiety, trauma history, biological predispositions, and social-environmental factors. These elements interact to create a condition where physical symptoms are experienced intensely and persistently, despite the absence of a clear medical explanation. Understanding this complex causation helps guide effective treatment approaches that address both mind and body.